Publications:

Hydrophobic interactions between the S5 segment and the pore helix stabilizes the closed state of Slo2.1 potassium channels.

Biochimica Et Biophysica Acta

Suzuki, Tomoyuki T; Hansen, Angela A; Sanguinetti, Michael C MC

Publication Date: 2016-04

Variant appearance in text: KCNQ1: V310A

PubMed Link: 26724206

Variant Present in the following documents:

• Main text

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Tuning of EAG K(+) channel inactivation: molecular determinants of amplification by mutations and a small molecule.

The Journal Of General Physiology

Garg, Vivek V; Sachse, Frank B FB; Sanguinetti, Michael C MC

Publication Date: 2012-09

Variant appearance in text: KCNQ1: V310A

PubMed Link: 22930803

Variant Present in the following documents:

• Main text
• JGP_201210826.pdf

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Allosteric features of KCNQ1 gating revealed by alanine scanning mutagenesis.

Biophysical Journal

Ma, Li-Juan LJ; Ohmert, Iris I; Vardanyan, Vitya V

Publication Date: 2011-02-16

Variant appearance in text: KCNQ1: V310A

PubMed Link: 21320432

Variant Present in the following documents:

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Mutation of colocalized residues of the pore helix and transmembrane segments S5 and S6 disrupt deactivation and modify inactivation of KCNQ1 K+ channels.

The Journal Of Physiology

Seebohm, Guiscard G; Westenskow, Peter P; Lang, Florian F; Sanguinetti, Michael C MC

Publication Date: 2005-03-01

Variant appearance in text: KCNQ1: V310A

PubMed Link: 15649981

Variant Present in the following documents:

• Main text

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